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claudio.talora@uniroma1.it
Claudio Talora
Professore Ordinario
Struttura:
DIPARTIMENTO DI MEDICINA MOLECOLARE
E-mail:
claudio.talora@uniroma1.it
Pagina istituzionale corsi di laurea
Curriculum Sapienza
Pubblicazioni
Titolo
Pubblicato in
Anno
Mir-34a-5p Mediates Cross-Talk between M2 Muscarinic Receptors and Notch-1/EGFR Pathways in U87MG Glioblastoma Cells: Implication in Cell Proliferation
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
2018
Yeast-Based Screen to Identify Natural Compounds with a Potential Therapeutic Effect in Hailey-Hailey Disease
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
2018
Autophagy processes are dependent on EGF receptor signaling
ONCOTARGET
2018
Identification of a novel chalcone derivative that inhibits Notch signaling in T-cell acute lymphoblastic leukemia
SCIENTIFIC REPORTS
2017
Maml1 acts cooperatively with Gli proteins to regulate Sonic hedgheog signaling pathway
CELL DEATH & DISEASE
2017
Prolyl-isomerase Pin1 controls Notch3 protein expression and regulates T-ALL progression
ONCOGENE
2016
Glutathione S-transferase ϴ-subunit as a phenotypic suppressor of pmr1Δ strain, the Kluyveromyces lactis model for Hailey-Hailey disease
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
2016
A threshold level of NFATc1 activity facilitates thymocyte differentiation and opposes notch-driven leukaemia development
NATURE COMMUNICATIONS
2016
Regulation of proapoptotic proteins Bak1 and p53 by miR-125b in an experimental model of Alzheimer's disease: Protective role of 17β-estradiol
NEUROSCIENCE LETTERS
2016
In vitro toxicity studies of zinc oxide nano- and microrods on mammalian cells: A comparative analysis
MATERIALS LETTERS
2016
The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease
SCIENTIFIC REPORTS
2016
The deregulated expression of miR-125b in acute myeloid leukemia is dependent on the transcription factor C/EBPalpha
LEUKEMIA
2015
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Progetti di Ricerca
Oxidative-stress and Notch link in keratinocyte transformation and differentiation.
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