Valeria Antonietta Pietropaolo

Pubblicazioni

Titolo	Pubblicato in	Anno
Differentiation of highly pathogenic strains of human JC polyomavirus in neurological patients by next generation sequencing	JOURNAL OF CLINICAL VIROLOGY	2024
Merkel Cell Polyomavirus in the Context of Oral Squamous Cell Carcinoma and Oral Potentially Malignant Disorders	BIOMEDICINES	2024
High rates of anal Merkel Cell Polyomavirus and HPV co‐infection among people living with HIV	JOURNAL OF MEDICAL VIROLOGY	2024
Regulation of transcriptional activity of Merkel cell polyomavirus large T-Antigen by PKA-mediated phosphorylation	INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES	2023
JC polyomavirus. A short review of its biology, its association with progressive multifocal leukoencephalopathy, and the diagnostic value of different methods to manifest its activity or presence	EXPERT REVIEW OF MOLECULAR DIAGNOSTICS	2023
Detection of human neurotropic JCPyV DNA sequence in pediatric anaplastic xanthoastrocytoma	JOURNAL OF NEUROVIROLOGY	2023
Detection of Merkel cell polyomavirus (mcpyv) dna and transcripts in Merkel cell carcinoma (mcc)	PATHOGENS	2023
Phosphorylation of human polyomavirus large and small t antigens. An ignored research field	VIRUSES	2023
Diagnostic Value of JC Polyomavirus Viruria, Viremia, Serostatus and microRNA Expression in Multiple Sclerosis Patients Undergoing Immunosuppressive Treatment	JOURNAL OF CLINICAL MEDICINE	2022
Functional Domains of the Early Proteins and Experimental and Epidemiological Studies Suggest a Role for the Novel Human Polyomaviruses in Cancer.	FRONTIERS IN MICROBIOLOGY	2022
Prevalence of MCPyV, HPyV6, HPyV7 and TSPyV in Actinic Keratosis Biopsy Specimens.	VIRUSES	2022
The inhibition of DNA viruses by the amphibian antimicrobial peptide temporin G. A virological study addressing HSV-1 and JPCyV	INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES	2022
COS-7 and SVGp12 Cellular Models to Study JCPyV Replication and MicroRNA Expression after Infection with Archetypal and Rearranged-NCCR Viral Strains	VIRUSES	2022
High prevalence of Merkel cell polyomavirus is associated with dysregulation in transcript levels of TLR9 and type I IFNs in a large cohort of CF patients from the Italian (Lazio) reference center for cystic fibrosis	MICROBIAL PATHOGENESIS	2022
Common microbial genital infections and their impact on the innate immune response to HPV in cervical cells	PATHOGENS	2022
Detection analysis and study of genomic region variability of JCPyV, BKPyV, MCPyV, HPyV6, HPyV7 and QPyV in the urine and plasma of HIV-1-infected patients	VIRUSES	2022
Reply to Henriksen, S.; Rinaldo, C.H. Should SVGp12 be used for JC polyomavirus studies? comment on “prezioso et al. COS-7 and SVGp12 cellular models to study JCPyV replication and microRNA expression after infection with archetypal and rearranged-NCCR viral strains. Viruses 2022, 14, 2070	VIRUSES	2022
SARS-CoV-2 Entry Genes Expression in Relation with Interferon Response in Cystic Fibrosis Patients	MICROORGANISMS	2021
Evaluation of Merkel Cell Polyomavirus DNA in Tissue Samples from Italian Patients with Diagnosis of MCC.	VIRUSES	2021
HPyV6 and HPyV7 in urine from immunocompromised patients.	VIROLOGY JOURNAL	2021

ERC

LS6_5
LS7
LS7_9

Progetti di Ricerca

Study of the Polyomavirus JC microRNAs circulating in biological fluids during viral persistence among relapsing-remitting multiple sclerosis patients treated with Disease Modifying Therapies.
Development of in vitro lung cells and organoid cultures to offer remarkable new model systems to study Merkel Cell Polyomavirus molecular biology and oncogenic mechanisms involved in malignancies other than Merkel Cell Carcinoma.
Development of in vitro model for the human Polyomavirus JC replication: analysis of rearrangements within NCCR structural organization during infection.
A more accurate risk biomarkers recognition for Progressive Multifocal Leukoencephalopathy (PML) caused by JC virus in patients with multiple sclerosis during treatment with disease-modifying therapies (DMTs): an ongoing clinical challenge.
IN VIVO STUDY OF MERKEL CELL POLYOMAVIRUS AS A TRIGGER OF EXACERBATION IN CYSTIC FIBROSIS PATIENTS AND DEVELOPMENT OF AN IN VITRO CULTURE MODEL BASED ON PRIMARY BRONCHIAL/TRACHEAL EPITHELIAL CELLS.