Titolo |
Pubblicato in |
Anno |
The homeobox transcription factor MEIS2 is a regulator of cancer cell survival and IMiDs activity in multiple myeloma. modulation by bromodomain and Extra-Terminal (BET) protein inhibitors |
CELL DEATH & DISEASE |
2019 |
Activation of liver X receptor up-regulates the expression of the NKG2D ligands MICA and MICB in multiple myeloma through different molecular mechanisms |
THE FASEB JOURNAL |
2019 |
The POU-Domain Transcription Factor Oct-6/POU3F1 as a Regulator of Cellular Response to Genotoxic Stress |
CANCERS |
2019 |
Innate Immune Response Regulation by the Human RNASET2 Tumor Suppressor Gene |
FRONTIERS IN IMMUNOLOGY |
2019 |
NKG2D and its ligands: one for all, all for one |
FRONTIERS IN IMMUNOLOGY |
2018 |
Drug-induced senescent Multiple Myeloma cells elicit NK cell proliferation by direct or exosome-mediated IL-15 trans-presentation |
CANCER IMMUNOLOGY RESEARCH |
2018 |
MICA-129 dimorphism and soluble MICA are associated with the progression of multiple myeloma |
FRONTIERS IN IMMUNOLOGY |
2018 |
Translating the anti-myeloma activity of Natural Killer cells into clinical application |
CANCER TREATMENT REVIEWS |
2018 |
Key role of the CD56lowCD16low Natural Killer cell subset in the recognition and killing of Multiple Myeloma cells |
CANCERS |
2018 |
p38 MAPK differentially controls NK activating ligands at transcriptional and post-transcriptional level on multiple myeloma cells |
ONCOIMMUNOLOGY |
2017 |
Genotoxic stress modulates the release of exosomes from multiple myeloma cells capable of activating NK cell cytokine production: role of HSP70/TLR2/NF-kB axis |
ONCOIMMUNOLOGY |
2017 |
Innate immune activating ligand SUMOylation affects tumor cell recognition by NK cells |
SCIENTIFIC REPORTS |
2017 |
Natural killer cell response to chemotherapy-stressed cancer cells: Role in tumor immunosurveillance. |
FRONTIERS IN IMMUNOLOGY |
2017 |
Immunoregulatory and effector activities of nitric oxide and reactive nitrogen species in cancer |
CURRENT MEDICINAL CHEMISTRY |
2016 |
Inhibition of bromodomain and extra-terminal (BET) proteins increases NKG2D ligand MICA expression and sensitivity to NK cell-mediated cytotoxicity in multiple myeloma cells. role of cMYC-IRF4-miR-125b interplay |
JOURNAL OF HEMATOLOGY & ONCOLOGY |
2016 |
Distinct Roles for Human Cytomegalovirus Immediate Early Proteins IE1 and IE2 in the Transcriptional Regulation of MICA and PVR/CD155 Expression |
JOURNAL OF IMMUNOLOGY |
2016 |
Targeting NKG2D and NKp30 ligands shedding to improve NK cell–based immunotherapy |
CRITICAL REVIEWS IN IMMUNOLOGY |
2016 |
NKG2D and DNAM-1 Ligands: Molecular Targets for NK Cell-Mediated Immunotherapeutic Intervention in Multiple Myeloma |
BIOMED RESEARCH INTERNATIONAL |
2015 |
The IMiDs targets IKZF-1/3 and IRF4 as novel negative regulators of NK cell-activating ligands expression in multiple myeloma |
ONCOTARGET |
2015 |
Ubiquitin-dependent endocytosis of NKG2D-DAP10 receptor complexes activates signaling and functions in human NK cells |
SCIENCE SIGNALING |
2015 |