| Titolo |
Pubblicato in |
Anno |
| Early and selective activation and subsequent alterations to the unfolded protein response in down syndrome mouse models |
JOURNAL OF ALZHEIMER'S DISEASE |
2018 |
| Disturbance of redox homeostasis in down syndrome: role of iron dysmetabolism |
FREE RADICAL BIOLOGY & MEDICINE |
2018 |
| Poly-ubiquitin profile in Alzheimer disease brain |
NEUROBIOLOGY OF DISEASE |
2018 |
| Proteomic identification of altered protein O-GlcNAcylation in a triple transgenic mouse model of Alzheimer's disease |
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE |
2018 |
| Biliverdin reductase-A impairment links brain insulin resistance with increased Aβ production in an animal model of aging: implications for Alzheimer disease |
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE |
2018 |
| Protein nitration profile of CD3+ lymphocytes from Alzheimer disease patients: novel hints on immunosenescence and biomarker detection |
FREE RADICAL BIOLOGY & MEDICINE |
2018 |
| Intranasal rapamycin ameliorates Alzheimer-like cognitive decline in a mouse model of Down syndrome |
TRANSLATIONAL NEURODEGENERATION |
2018 |
| The triangle of death in Alzheimer's disease brain: the aberrant cross-talk among energy metabolism, mammalian target of rapamycin signaling, and protein homeostasis revealed by redox proteomics |
ANTIOXIDANTS & REDOX SIGNALING |
2017 |
| Polyubiquitinylation profile in down syndrome brain before and after the development of Alzheimer neuropathology |
ANTIOXIDANTS & REDOX SIGNALING |
2017 |
| HNE-modified proteins in Down syndrome: involvement in development of Alzheimer disease neuropathology |
FREE RADICAL BIOLOGY & MEDICINE |
2017 |
| Modulation of GLP-1 signaling as a novel therapeutic approach in the treatment of Alzheimer’s disease pathology |
EXPERT REVIEW OF NEUROTHERAPEUTICS |
2017 |
| Increased mammalian target of rapamycin signaling contributes to the accumulation of protein oxidative damage in a mouse model of down's syndrome |
NEURODEGENERATIVE DISEASES |
2016 |
| Impairment of biliverdin reductase-a promotes brain insulin resistance in Alzheimer disease: a new paradigm |
FREE RADICAL BIOLOGY & MEDICINE |
2016 |
| Activation of p53 in down syndrome and in the ts65dn mouse brain is associated with a pro-apoptotic phenotype |
JOURNAL OF ALZHEIMER'S DISEASE |
2016 |
| It is all about (U)biquitin: role of altered ubiquitin-proteasome system and UCHL1 in Alzheimer disease |
OXIDATIVE MEDICINE AND CELLULAR LONGEVITY |
2016 |
| Editorial: Oxidative stress and Alzheimer disease: where do we stand? |
CURRENT ALZHEIMER RESEARCH |
2016 |
| Aberrant protein phosphorylation in Alzheimer disease brain disturbs pro-survival and cell death pathways |
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE |
2016 |
| Bach1 overexpression in down syndrome correlates with the alteration of the ho-1/bvr-a system: insights for transition to Alzheimer's disease |
JOURNAL OF ALZHEIMER'S DISEASE |
2015 |
| Strategy to reduce free radical species in Alzheimer's disease: an update of selected antioxidants |
EXPERT REVIEW OF NEUROTHERAPEUTICS |
2015 |
| Basal brain oxidative and nitrative stress levels are finely regulated by the interplay between superoxide dismutase 2 and p53 |
JOURNAL OF NEUROSCIENCE RESEARCH |
2015 |
