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eugenio.barone@uniroma1.it
Eugenio Barone
Professore Ordinario
Struttura:
DIPARTIMENTO DI SCIENZE BIOCHIMICHE "ALESSANDRO ROSSI FANELLI"
E-mail:
eugenio.barone@uniroma1.it
Pagina istituzionale corsi di laurea
Curriculum Sapienza
Pubblicazioni
Titolo
Pubblicato in
Anno
Early and selective activation and subsequent alterations to the unfolded protein response in down syndrome mouse models
JOURNAL OF ALZHEIMER'S DISEASE
2018
Disturbance of redox homeostasis in down syndrome: role of iron dysmetabolism
FREE RADICAL BIOLOGY & MEDICINE
2018
Poly-ubiquitin profile in Alzheimer disease brain
NEUROBIOLOGY OF DISEASE
2018
Proteomic identification of altered protein O-GlcNAcylation in a triple transgenic mouse model of Alzheimer's disease
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE
2018
Biliverdin reductase-A impairment links brain insulin resistance with increased Aβ production in an animal model of aging: implications for Alzheimer disease
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE
2018
Protein nitration profile of CD3+ lymphocytes from Alzheimer disease patients: novel hints on immunosenescence and biomarker detection
FREE RADICAL BIOLOGY & MEDICINE
2018
Intranasal rapamycin ameliorates Alzheimer-like cognitive decline in a mouse model of Down syndrome
TRANSLATIONAL NEURODEGENERATION
2018
The triangle of death in Alzheimer's disease brain: the aberrant cross-talk among energy metabolism, mammalian target of rapamycin signaling, and protein homeostasis revealed by redox proteomics
ANTIOXIDANTS & REDOX SIGNALING
2017
Polyubiquitinylation profile in down syndrome brain before and after the development of Alzheimer neuropathology
ANTIOXIDANTS & REDOX SIGNALING
2017
HNE-modified proteins in Down syndrome: involvement in development of Alzheimer disease neuropathology
FREE RADICAL BIOLOGY & MEDICINE
2017
Modulation of GLP-1 signaling as a novel therapeutic approach in the treatment of Alzheimer’s disease pathology
EXPERT REVIEW OF NEUROTHERAPEUTICS
2017
Increased mammalian target of rapamycin signaling contributes to the accumulation of protein oxidative damage in a mouse model of down's syndrome
NEURODEGENERATIVE DISEASES
2016
Impairment of biliverdin reductase-a promotes brain insulin resistance in Alzheimer disease: a new paradigm
FREE RADICAL BIOLOGY & MEDICINE
2016
Activation of p53 in down syndrome and in the ts65dn mouse brain is associated with a pro-apoptotic phenotype
JOURNAL OF ALZHEIMER'S DISEASE
2016
It is all about (U)biquitin: role of altered ubiquitin-proteasome system and UCHL1 in Alzheimer disease
OXIDATIVE MEDICINE AND CELLULAR LONGEVITY
2016
Editorial: Oxidative stress and Alzheimer disease: where do we stand?
CURRENT ALZHEIMER RESEARCH
2016
Aberrant protein phosphorylation in Alzheimer disease brain disturbs pro-survival and cell death pathways
BIOCHIMICA ET BIOPHYSICA ACTA. MOLECULAR BASIS OF DISEASE
2016
Bach1 overexpression in down syndrome correlates with the alteration of the ho-1/bvr-a system: insights for transition to Alzheimer's disease
JOURNAL OF ALZHEIMER'S DISEASE
2015
Strategy to reduce free radical species in Alzheimer's disease: an update of selected antioxidants
EXPERT REVIEW OF NEUROTHERAPEUTICS
2015
Basal brain oxidative and nitrative stress levels are finely regulated by the interplay between superoxide dismutase 2 and p53
JOURNAL OF NEUROSCIENCE RESEARCH
2015
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Progetti di Ricerca
ABERRANT INSULIN SIGNALING CONTRIBUTES TO DEVELOPMENT OF ALZHEIMER DISEASE: SEARCH FOR NOVEL THERAPEUTIC STRATEGIES
Biliverdin reductase-A impairment mediates the development of insulin resistance in obesity and T2DM: a novel mechanism of action.
Unravelling a novel mechanism favoring brain insulin resistance development and its impact on Alzheimer disease neuropathology
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