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lucia.dimarcotullio@uniroma1.it
Lucia Di Marcotullio
Professore Ordinario
Struttura:
DIPARTIMENTO DI MEDICINA MOLECOLARE
E-mail:
lucia.dimarcotullio@uniroma1.it
Pagina istituzionale corsi di laurea
Curriculum Sapienza
Pubblicazioni
Titolo
Pubblicato in
Anno
Hypomorphic Recessive Variants in SUFU Impair the Sonic Hedgehog Pathway and Cause Joubert Syndrome with Cranio-facial and Skeletal Defects
AMERICAN JOURNAL OF HUMAN GENETICS
2017
Targeting Gli acetylation in Shh-medulloblastoma: A successful preclinical approach
INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
2017
Corrigendum: Selective targeting of HDAC1/2 elicits anticancer effects through Gli1 acetylation in preclinical models of SHH Medulloblastoma
SCIENTIFIC REPORTS
2017
Nuclear Factor of Activated T Cells-dependent Down-regulation of the Transcription Factor Glioma-associated Protein 1 (GLI1) Underlies the Growth Inhibitory Properties of Arachidonic Acid
THE JOURNAL OF BIOLOGICAL CHEMISTRY
2016
The energy sensor AMPK regulates Hedgehog signaling in human cells through a unique Gli1 metabolic checkpoint
ONCOTARGET
2016
MK-4101 - a potent inhibitor of the hedgehog pathway - is highly active against medulloblastoma and basal cell carcinoma
MOLECULAR CANCER THERAPEUTICS
2016
Inhibition of Hedgehog-dependent tumors and cancer stem cells by a newly identified naturally occurring chemotype
CELL DEATH & DISEASE
2016
SCF (Fbxl17) ubiquitylation of Sufu regulates Hedgehog signaling and medulloblastoma development
EMBO JOURNAL
2016
Gli1/DNA interaction is a druggable target for Hedgehog-dependent tumors
EMBO JOURNAL
2015
Digging a hole under Hedgehog: Downstream inhibition as an emerging anticancer strategy
BIOCHIMICA ET BIOPHYSICA ACTA-REVIEWS ON CANCER
2015
The MRN complex is transcriptionally regulated by MYCN during neural cell proliferation to control replication stress
CELL DEATH AND DIFFERENTIATION
2015
Non-canonical Hedgehog/ampk-mediated control of polyamine metabolism supports neuronal and medulloblastoma cell growth
DEVELOPMENTAL CELL
2015
New Indole Tubulin Assembly Inhibitors Cause Stable Arrest of Mitotic Progression, Enhanced Stimulation of Natural Killer Cell Cytotoxic Activity, and Repression of Hedgehog-Dependent Cancer
JOURNAL OF MEDICINAL CHEMISTRY
2015
Insights into gli factors ubiquitylation methods
Methods in Molecular Biology
2015
Targeting GLI factors to inhibit the Hedgehog pathway
TRENDS IN PHARMACOLOGICAL SCIENCES
2015
Cullin3-BTB interface: a novel target for stapled peptides
PLOS ONE
2015
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Progetti di Ricerca
Oncogenic role of the aminopeptidase ERAP1 in Hedgehog-dependent cancer
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