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fabio.didomenico@uniroma1.it
Fabio Di Domenico
Professore Ordinario
Struttura:
DIPARTIMENTO DI SCIENZE BIOCHIMICHE "ALESSANDRO ROSSI FANELLI"
E-mail:
fabio.didomenico@uniroma1.it
Pagina istituzionale corsi di laurea
Curriculum Sapienza
Pubblicazioni
Titolo
Pubblicato in
Anno
Oxidative signature of cerebrospinal fluid from mild cognitive impairment and Alzheimer disease patients
FREE RADICAL BIOLOGY & MEDICINE
2016
Bach1 overexpression in down syndrome correlates with the alteration of the ho-1/bvr-a system: insights for transition to Alzheimer's disease
JOURNAL OF ALZHEIMER'S DISEASE
2015
Strategy to reduce free radical species in Alzheimer's disease: an update of selected antioxidants
EXPERT REVIEW OF NEUROTHERAPEUTICS
2015
Pharmacologic approaches against advanced glycation end products (ages) in diabetic cardiovascular disease
RESEARCH IN CARDIOVASCULAR MEDICINE
2015
Basal brain oxidative and nitrative stress levels are finely regulated by the interplay between superoxide dismutase 2 and p53
JOURNAL OF NEUROSCIENCE RESEARCH
2015
Redox proteomics in human biofluids: sample preparation, separation and immunochemical tagging for analysis of protein oxidation
Systems Biology of Alzheimer's Disease
2015
Aberrant insulin signaling in Alzheimer's disease: current knowledge
FRONTIERS IN NEUROSCIENCE
2015
MTOR signaling in aging and neurodegeneration: At the crossroad between metabolism dysfunction and impairment of autophagy
NEUROBIOLOGY OF DISEASE
2015
Alteration of mTOR signaling occurs early in the progression of Alzheimer disease (AD): analysis of brain from subjects with pre-clinical AD, amnestic mild cognitive impairment and late-stage AD
JOURNAL OF NEUROCHEMISTRY
2015
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Progetti di Ricerca
Deciphering the the interplay between the unfolded protein response and insulin resistance in Down syndrome brain
Inhibition of PERK pathway for the early treatment of Alzheimer Disease-like cognitive decline in Down Syndrome
The sweet link between aberrant brain metabolism and cognitive decline: A novel role for altered protein O-GlcNAcylation.
Deciphering the role of aberrant PERK/NRF2 axis in Down Syndrome
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